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Importantly, these characteristic mitochondrial functional abnormalities have been demonstrated in human ASD studies (summarised in Table 2) and reflect a generally reduced mitochondrial respiration and functional capacity
Low levels of this important antioxidant are common in people suffering from chronic fatigue, autoimmune issues and cardiovascular disease
Mechanical breaking of microtubules in axons during dynamic stretch injury underlies delayed elasticity, microtubule disassembly and axon degeneration
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