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It would remain the responsibility of Primary Care to diagnose and treat B12 deficient patients but they would have to choose to
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provided evidence of B cell ferroptosis in SLE patients and SLE mouse models, suggesting that ferroptosis may regulate B cell differentiation and plasma cell formation, thereby implicating this process in SLE pathogenesis [102]
Sci Rep 10(1):2259 Lora J, Alonso FJ, Segura JA, Lobo C, Mrquez J, Mats JM (2004) Antisense glutaminase inhibition decreases glutathione antioxidant capacity and increases apoptosis in Ehrlich ascitic tumour cells
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