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Results Senescence-associated markers were enhanced in keloid fibroblasts and lesions Analysis of the heterogeneity of keloid cells revealed 28 cellular clusters (027) using unsupervised clustering of 34,043 cells based on established lineage-specific marker genes (Supplementary Fig

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Chronic tendinopathy is characterized by failed healing response, disorganized collagen matrix (tendinosis), reduced tenocyte activity, and inadequate vascularization, all of which align directly with BPC-157's documented mechanisms of fibroblast stimulation, collagen remodeling, and angiogenesis

The pattern fades by 1-2 hours