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However, in highly invasive breast cancer cell lines MDA-MB-231 and MDA-MB-468, high concentrations (100 nM) of liraglutide promote breast cancer progression by activating GLP-1R via the NOX4/ROS/VEGF signaling pathway [18]
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This process can generate free radicals and produce ROS under the action of endogenous substances in the cell, thereby achieving the purpose of local killing of the tumor
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