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The mechanistic basis for TB-500s neural repair activity is the expression of Thymosin Beta-4 in neuronal and glial cells and its role in regulating the actin dynamics that underlie neural cell migration, axonal growth cone advance, and the oligodendrocyte precursor cell migration toward demyelinated lesion sites that initiates remyelination
[7] Population pharmacokinetic modeling reveals a biphasic disposition (distribution half-life 15-30 minutes
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