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50 found that tubuloside A could alleviate nonsteroidal anti-inflammatory drug-induced hepatorenal oxidative injury via the Nrf2/HO-1 pathway, involving changes in the mRNA expression of genes Nrf2, HO-1, NQO1, IL-6, iNOS, COX-2, TNF-, IL-1, and NF-B, as well as apoptotic processes (Bcl-2, caspase-3, and Bax)

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