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Of note, E138 1.33b R in GLP-1R and R131 1.33b E in GIPR modestly increased and decreased the potency of retatrutide, respectively, indicating the receptor-specific adaptability of peptide recognition at this site (Fig

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[4] enhanced cell migration, where MSCs respond to chemotactic signals from the injury site and pro-migratory factors secreted by themselves, thereby homing to the skin wound area and participating in wound repair alongside KCs, macrophages, and endothelial cells migrating to the wound tissue [125, 126]