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ROS-induced NO inactivation can impair vascular endothelial function, contributing to various pathologies like atherosclerosis, hypertension, cardiovascular disease, and central nervous system disorders (27, 28)
These alterations were accompanied by increased expression of Cas-3 and NF-B, elevated levels of pro-inflammatory cytokines (TNF-, IL-6), and significant oxidative stress, as evidenced by increased MDA and decreased antioxidant enzyme levels (SOD, CAT, GSH-Px)
Biochem Pharmacol 36: 3169-3173
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