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Alternatively, GSH could have potentially prevented NRF2 activation through both KEAP1-independent mechanisms
Femiano F, Lanza A, Buonaiuto C, et al
Johnson 5 , (1)Department of Medicine, George Washington University Hospital, Washington, DC, (2)Alexandria University Faculty of Medicine, Alexandria, Egypt, (3)The George Washington University School of Medicine and Health Sciences, (4)George Washington University Medical Faculty Associates, (5)Department of Surgery, George Washington University Medical Faculty Associates, (6)Department of Endocrinology, George Washington University Medical Faculty Associates Background: Nonalcoholic fatty liver disease (NAFLD) is prevalent in up to 60% of patients with type 2 diabetes mellitus (T2DM)
