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Only through these approaches can the current gapclear mechanisms but poor clinical translationbe overcome, advancing ferroptosis-targeted therapy as a strategy to improve neurological outcomes in TBI patients

BPC-157's VEGFR2 engagement provides growth factor receptor signalling, while TB-500's actin-binding properties facilitate cytoskeletal remodelling necessary for cellular motility responses

[125] Norrish AE, Skeaff CM, Arribas GL, et al

Altered innate immune gene expression in LRRK2 knockout macrophages is driven by mitochondrial stresses, such as oxidative stress from low levels of purine metabolites and Drp1-dependent mitochondrial fragmentation