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To evaluate the role of TGF signalling in high glucose-induced PSC activation, we treated cells in the low-glucose group with 5 ng/mL TGF1 and cells in the high-glucose group with 10 M of the TGF pathway inhibitor SB431542
By stimulating GLUT4 translocation to the cell surface, resistance training improves glucose disposal and metabolic flexibilityeven without pharmacological intervention
Watkins JB, III
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