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(PubMed) Kutlu O
0.4 nmol and 4.0 nmol) of peptide treatments
As the dominant G-actin sequestering protein in mammalian cells, TB-500 binds globular actin with high affinity (Kd ~0.5 M) through a sophisticated triple-contact mechanism involving Lys-3, Lys-18, and Lys-38 residues, preventing spontaneous actin polymerization while maintaining a readily mobilizable reservoir for rapid cytoskeletal reorganization
The process is not as simple as stopping one and starting the other at what you think is equivalent
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