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MiR-520h inhibits viability and facilitates apoptosis of KGN cells through modulating IL6R and the JAK/STAT pathway
The expected benefits on heart disease, stroke and other cardiovascular and most kidney diseases are clearly seen
It is evident that CNS GLP-1R signaling is essential for mediating the pharmacological action of GLP-1R agonists on energy metabolism as genetic deletion of CNS GLP-1R signaling in mice ablates the action of peripherally administered liraglutide on food intake and body weight reduction (Sisley et al., The hypothalamus has also been implicated as a major center for mediating the pharmacological effects of GLP-1R action on energy metabolism
Lancet 392, 15191529
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