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The unfolded protein response protects human tumor cells during hypoxia through regulation of the autophagy genes MAP1LC3B and ATG5
KEGG pathway enrichment analysis was conducted to identify the potential signaling pathways associated with potential targets, and the analysis uncovered several significant pathways 15 (Fig
Chronic mitochondrial inefficiency is an established source of continuous reactive oxygen species generation and can further compromise GSH synthesis and regeneration through effects on ATP production and the -glutamyl cycle [38]
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