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Additionally, D3T upregulates silent information regulator 1 (SIRT1), Nrf2, and HO-1, ameliorating cognitive deficits in the Tg2576 AD mouse model ( D3T exhibits neuroprotective effects in neuroinflammation and IS, suggesting its potential as a treatment option for ferroptosis-related diseases

While specific therapies for IBM are still lacking, NAD + boosters have shown promising results in various models, including nematodes with amyloid aggregation, aged rodents, and human muscle cells from IBM patients