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Additional literature suggests that peptide-mediated regulation of cytokines and chemokines shapes the directional movement of immune populations toward injured sites, enabling coordinated repair rather than uncontrolled inflammation

This process involves a complex pathological network encompassing disrupted iron homeostasis, GPX4 inactivation, abnormal lipid metabolism, mitochondrial dysfunction, and amplified immune-inflammatory responses

TREM2 agonists, such as AL002, promote microglial transformation toward the anti-inflammatory M2 phenotype, thereby reducing the release of pro-inflammatory M1 cytokines (e.g., TNF-, IL-1) and indirectly mitigating inflammation-driven ferroptotic cascades

Restivo I, Attanzio A, Tesoriere L, Allegra M