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The adoption of a pro-inflammatory phenotype in microglia can be driven by various factors, including the presence of pro-inflammatory mediators, infiltration of peripheral immune cells, pathological deposits (such as tau aggregates), and viral proteins [e.g., proteins trans-activator of transcription (Tat)] [26, 27]
However, symptoms that resemble low blood sugar can still occur, especially early in treatment or when nutrition is inconsistent
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