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Other studies performed in mice demonstrate that pancreatic Y1-R activation facilitates the trans-differentiation of -cells into -cells, thereby improving insulin sensitivity (Lafferty, Flatt et al
Taken together, the above-mentioned data suggest that GSH might serve as an important wound signal that triggers wound-induced systemic [Ca 2+ ] cyt transmission, activates JA biosynthesis, and regulates plant defense responses
Zander M, Madsbad S, Madsen JL, Holst JJ (2002) Effect of 6-week course of glucagon-like peptide 1 on glycaemic control, insulin sensitivity, and beta-cell function in type 2 diabetes: a parallel-group study
Dietitian's Recommendation 5
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