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In particular, the glutamate-generating metabolism seems essential, as shown by a complete depletion of Gln during H
This built-in transience is exactly what pharmaceutical chemists have to fight against when they want a peptide to last
Interestingly, miR-99a-overexpressing human keratinocyte cells showed slower proliferation as compared to the control cells suggesting that miR-99a may function in the development of psoriasis by targeting IGF-1R
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