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The liver is the main site of ApoE synthesis and secretion, and the ApoE gene, especially the 4 allele, is the strongest genetic risk factor for sporadic AD, and some of the AD disease markers and therapeutic effects are even dependent on differences in ApoE genotype [82, 83]

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The more well known and researched ones are: [2] HLA-DR3, associated with thyroid gland atrophy HLA-DR5, associated with an enlarged thyroid gland These immune regulatory genes are closely associated with the autoimmune process of Hashimotos

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