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Chang, W.-C
Research Context & Areas of Scientific Interest In laboratory and preclinical settings, GHK-Cu has been studied for its involvement in: Copper-mediated biochemical pathways Cellular signaling and regulatory mechanisms Gene expression models in controlled environments Extracellular matrix interaction dynamics Molecular modeling involving peptide-metal complexes Cellular response behavior in in-vitro systems These observations originate from preclinical and laboratory research only and do not represent clinical or cosmetic effects in humans
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As do other arsenicals, it induces G 2 /M cell cycle arrest and triggers apoptosis through disruption of mitochondrial functions and JNK activation and is responsible for reactive oxygen species production (Garnier et al., 2013)
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