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These KPFs include genetic predisposition and injury triggered by viral infections/toxins and/or niche abnormalities, with inflammatory and immune dysfunction, leading to obliteration and eventual fibrosis and atresia of the bile ducts (26)
A fragment does not automatically have the same effects or safety profile as the whole
The research rationale for a selective NNMT inhibitor is to interrupt exactly that diversion, so the cell keeps more of its NAD+ pool and can run its mitochondrial machinery again
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