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Ahmed, Z., Wang, Y., Ahmad, A., Khan, S

Abstract Glucose-6-phosphate dehydrogenase (G6PD) deficiency, one of the most common human genetic enzymopathies, is caused by over 160 different point mutations and contributes to the severity of many acute and chronic diseases associated with oxidative stress, including hemolytic anemia and bilirubin-induced neurological damage particularly in newborns

This capacity allows them to colonize adhesion sites, including colonic crypts, goblet cells, and intestinal villi, in substantial numbers, thereby physically preventing pathogens from gaining a foothold and establishing infection (163, 192)

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