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Behaving as the terminal electron acceptor during cellular respiration, the high rate of O 2 electron flux is accompanied by an increased risk of ROS formation, which can induce irreversible damage to cellular organelles and their processes (Case 2017)
However, these benefits were reversible, with behaviors reverting toward baseline once the treatment stopped
They are well-documented
In these models, we take into account the reaction geometry of common thiol-disulfide exchange reactions and distinguish hypothetical protein areas that might either interact with the disulfide substrate (a scaffold site) or the reducing agent (an activator site) (Fig
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