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Although residues C32 and C88 of PfGrx are redox-inactive during the monothiol mechanism, they both facilitate the deglutathionylation of GSSR substrates
Our assay provides a more sensitive, specific, and flexible tool for use with human, mouse, and rat models in various states such as fasted and fed, glucose-stimulated insulin secretion, and diet-induced obesity, as well as perifusion.
These burps, medically termed sulphurous eructation, may potentially relate to the medication's effect on gastrointestinal motility, though the exact mechanism remains unconfirmed
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