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These MC1R polymorphism studies have contributed to understanding the molecular basis for the phenotypic consequences of loss-of-function MC1R variants in skin cancer risk and pigmentation biology
Secondly, significant weight loss and the resultant change in the inflammatory phenotype of obese adipose tissue would reduce the release into the circulation of key pro-fibrotic pro-inflammatory cytokines/adipokines such as TNF-, IL-6, leptin [31], visfatin and resistin, and increase levels of anti-fibrotic adipokines such as adiponectin [32]
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