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Senolytic FOXO4-DRI can inhibit FOXO4 binding by disrupting the p53-FOXO4 interaction and selectively cause apoptosis via p53 nuclear exclusion in senescent cells and aging mouse models, which may be a promising approach for preventing senescence 11
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Rifampicin reduced the bioluminescence of intracellular bacteria, consistent with its ability to penetrate mammalian cell membranes and inhibit bacterial transcription, thereby reducing metabolic activity (Fig
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