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Ferroptosis in chronic obstructive pulmonary disease: From cellular mechanisms to therapeutic application
Low dose cytosin arabinoside (LDCA) in chronic lymphocytic leukemia (CLL)
Furthermore, oxidative stress cascades involving ROS, neuroinflammatory pathways such as TLR-4, NFB, and NLRP3 inflammasome-driven pyroptosis, vascular endothelial growth factor signaling, as well as diverse forms of cell death, including organellar dysfunction, autophagy, apoptosis, ferroptosis, and necroptosis, require further research to assess their potential as effective therapeutic targets (130)
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